Using simultaneous recordings of electrophysiology, respiration, and resting-state fMRI, a respiration-associated neural network is identified.

Noninvasive human magnetoencephalography recordings characterize the functional relationship between respiration, neural oscillations, and performance in low-level perception.

Computational analyses of in-vivo cerebral activity show that gamma oscillations in the olfactory cortex originate from local feedback inhibition following each sniff and serve to segregate competing odor representations through a winner-take-all process, providing an optimal window to decode odor.

The bacterial pathogen Listeria monocytogenes requires respiration for the maintenance of cellular redox homeostasis.

Complex I of the mitochondrial respiratory chain is necessary for interferon gamma-mediated responses in antigen-presenting cells and is required for CD4+ T cell activation.

The primary rhythm generators controlling respiration are integrated in a much larger network of brain regions, including areas not typically considered to be respiratory control centers, and that allow respiration to be adapted to all forms of ongoing behavior.

The localization of respiratory complexes in the cell membrane emerges as a critical factor for controlling respiration in bacteria.

V1 damage impairs unconscious following movements of stimulus motion, even after conscious perception has been restored through psychophysical training, thus demonstrating distinct neural pathways for perception and action.

An unbiased description reveals potential implications for understanding the developmental mechanisms that match respiratory supply and demand during hypoxia.

Active expiration in anesthetized rats is driven by the lateral parafacial region, but requires an additional form of excitation from another source.