A novel behavioural paradigm reveals how neurons in the ventral hippocampus dynamically adapt their firing activity after the introduction of an anxiogenic location.

Input from the ventral hippocampus to the medial prefrontal cortex regulates social memory in wild type mice and atypically-strengthened input causes social memory dysfunction in Rett syndrome mice.

Optogenetics was used to reveal a novel role for the perirhinal cortex in regulating approach-avoidance behaviors under object-based motivational conflict.

In contrast to current models, inputs from midbrain limbic structures, but not from the hippocampus, are necessary for mammillary body contributions to memory.

A communication pathway linking memory processing and feeding behavior exists between the gut hormone ghrelin, the hippocampus and the hypothalamic neuropeptide orexin.

Combined optogenetics and electrophysiology in vivo identified the cellular substrate of hippocampal drive that initiates the oscillatory entrainment of prefrontal cortex in neonatal mice.

All-trans retinoic acid increases the number of excitatory synapses in the dorsal hippocampus of mice and improves the ability of neurons to express synaptopodin-dependent synaptic plasticity.

Two intrinsic factors converge to govern the differentiation and integration of distinct characteristics of the hippocampus in mice.

Self-motivated learning in the absence of external feedback can recruit the same brain network as reward-driven learning.

Amygdala input to ventral hippocampus is both excitatory and inhibitory, and the balance of these two projections controls the formation of place preference via differential innervation of each hippocampal output population.