GPi spiketrain feature analyses and clinical correlates of PD and dystonia.
(A) In dystonia, GPi neuronal output was slower, burstier, and more irregular compared to PD; however, (B) there were no significant differences in spiketrain oscillations. (C) In PD, increased power of low beta spiketrain oscillations was associated with greater symptom severity. In dystonia, lower neuronal firing rates, higher neuronal irregularity, and increased power of theta spiketrain oscillations were associated with greater symptom severity.