Figures and data
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Baseline characteristics of rats and resuscitation characteristics (x̄ ± s)
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Experimental design of the in-vivo study, Rats in all groups except the Sham group underwent cardiac arrest for 5 minutes and then received the corresponding intervention 10 minutes after ROSC. Four hours after ROSC, the 4-hour group was used to collect myocardial tissue and blood samples for detection, while the 72-hour group was used for survival detection. BL, baseline; CA, cardiac arrest; ROSC, return of spontaneous circulation.
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Observational results at 4 and 72 hours after cardiopulmonary resuscitation in rats. (A) Echocardiograms of rats in each group from baseline to 4 hours following ROSC (n=7). (B) EF of rats in each group from baseline to 4 hours following ROSC (n=7). (C) HR and MAP changes during post -ROSC in 4 hours (n=7). (D) Survival rate during the first 72 hours following ROSC (n=10). Myocardial function between groups was compared by time-based measurements in each group using repeated-measures ANOVA. The survival rate between groups was compared by the Kaplan-Meier survival analysis test. * P<0.05 vs. the Sham group and # P<0.05 vs. the Mito group. BL, baseline; EF, ejection fraction; HR, heart rate; MAP, mean arterial pressure; bpm, beats per minute; mmHg, millimeters of mercury; AMV, after mechanical ventilation; ROSC, return of spontaneous circulation.
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Assessment of the viability and purity of isolated mitochondria. (A) JC-1 staining of mitochondria after isolation from muscle. The staining of isolated mitochondria by JC-1 is visible either as red for J-aggregates or green for J-monomers. The intensity of the red color indicates that the isolated mitochondria had a high membrane potential, confirming their quality for transplantation. Scale bar = 100 µm. (B) SDS/PAGE analysis of fractions obtained during the purification of muscle mitochondria. GAPDH is only expressed in muscle, confirming its purity for transplantation.
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Localization and uptake of transplanted mitochondria in endocardium, myocardial tissue was stained for anti-α-actinin 2 (ACTN2; green) and nuclei (blue); the pre-stained isolated mitochondria were labeled red (n=3). Scale bar = 80 µm.
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Administration of mitochondria ameliorates ischemia reperfusion-mediated mitochondrial alterations in cardiomyocytes four hours after ROSC. (A and B) A and B represent the detection of fluorescence intensity of TOM20. A represents the image, while B represents the quantitative data. Scale bar = 100 µm (n = 3). (C and D) C shows representative photographs of mitochondrial morphology obtained through TEM examination, with arrows indicate calcium accumulation. D illustrates the degree of mitochondrial damage (n=3). Scale bars = 500 nm. (E and F) Changes in myocardial mitochondrial complex II and IV enzyme activities in hearts (n=7). (G) The ATP content in myocardial tissue was measured by colorimetry (n=7). (H and I) mPTP opening was detected by Calcein staining. I represent the quantitative analysis of the mean fluorescent intensity acquired in H (n = 3). Analyses were performed using ANOVA with Tukey’s post hoc test. mPTP opening detection between groups was compared by time-based measurements in each group using repeated-measures ANOVA. The data were expressed as the mean ± standard deviation (SD). * P<0.05 vs. the Sham group and # P<0.05 vs. the Mito group.
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Mitochondrial transplantation reduces myocardial damage 4 hours after ROSC. (A) Detection of mitochondrial ROS in various groups (n=3). (B and C) Changes in malondialdehyde and superoxide dismutase activity level in cardiac tissue (n=7). (D) Myocardial apoptosis level was examined using TUNEL (n=3). Scale bar = 100 µm. (E) The percentage of myocardial apoptosis was examined using flow cytometry (n=3). (F) Quantitative analysis of myocardial TUNEL apoptosis index and flow apoptosis rate (n=3). (G and H) Immunoblotting and quantitative analysis of the expression level of cleaved caspase-3 in the myocardium 4 hours after ROSC (n=3). (I and J) The changes in CK-MB and cTn-I level in the serum of rats were examined using ELISA (n=7). (K and L) Representative histological sections of the myocardium stained with hematoxylin and eosin. Myocardium from each experimental group were subjected to histological evaluation (n=3), Scale bar = 100 µm. Analyses were performed using ANOVA with Tukey’s post hoc test. The data were expressed as mean ± standard deviation (SD). * P < 0.05 vs. Sham group and # P < 0.05 vs. Mito group. CK-MB, creatine kinase-MB fraction; cTn-I: cardiac troponin-I.
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Exogenous mitochondrial transplantation improved cardiac function after CPR. The specific mechanism involved may be related to the improvement in mitochondrial function, thus reducing the oxidative-stress response and apoptosis of myocardial cells. These dates suggest possible advantage in mitochondrial transplantation following CPR.