A hybrid mouse/human model using mesenchymal progenitor cells reveals dynamics of human adipose tissue development and mechanisms that may enhance human adipose thermogenic capacity.

Combined fate mapping and genetic deletion studies reveal that PDGFRα+ cells and PDGFRα gene itself are required for adipose tissue development but not for adult tissue homeostasis.

Clock protein NR1D1 regulates white adipose tissue expansion and development of obesity-related pathology.

TAF7L acts as a molecular switch that regulates the development of mesenchymal stem cells.

The size of subcutaneous, but not visceral, fat depots is dependent on the number of embryonic adipocyte progenitors.

PDZ-RhoGEF controls adipose tissue abundance and whole body metabolism.

CD107a expression divides osteogenic from adipogenic perivascular precursor cells within human white adipose tissue.

The regulation of sympathetic innervation density by beige adipocytes in subcutaneous fat is important during a critical developmental window, but dispensable during adulthood.

Cell-cell interaction between neural crest-derived cells and mesoderm-derived cells determine cell fate of mesoderm-derived cell in tongue development through Hh signaling.

Identification of bone marrow Adipoq-lineage progenitors as a novel and major cellular source of M-CSF in mice and humans, which controls bone marrow macrophage development, osteoclastogenesis, and bone mass in physiological and pathological conditions.