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Ion conduction in the calcium-activated chloride channel TMEM16A is directly regulated by calcium, which binds to a site close to the pore thereby shaping the electrostatics at its intracellular entrance.

KChIP3 localisation by intracellular calcium oscillations controls mucin release from colonic goblet cells.

Potassium ions enter and leave human sperm cells via a calcium-dependent ion channel that is also pH-independent.

High-speed calcium imaging from the axon initial segment shows that sodium channels are in part permeable to calcium ions.

A transient receptor channel in Toxoplasma gondii that conducts calcium represents the first molecular element that initiates upstream calcium signals that activate parasite pathogenic pathways.

Goblet cells secrete mucins—which are key components of mucus—in a process that is regulated by calcium ions, which enter the goblet cells via a mechanism involving a channel protein called TRPM5.

Cytosolic S100A8/A9 enhances Ca²⁺ availability at LFA-1 adhesion sites, facilitating neutrophil recruitment and extravasation by supporting proper integrin signaling and cytoskeletal rearrangement.

NG2 glial cells respond to synaptic input with calcium signals in the absence of action potentials and process synaptic depolarizations with somatic and dendritic voltage gated channels.

Soluble amyloid-β precursor peptide has no functional effects at recombinant and native GABA_B receptors.

Live-cell imaging and biochemical studies reveal that bradyzoites exhibit reduced Ca²⁺ stores, dampened calcium responses, and reduced energy levels consistent with their quiescent sate, but that they rapidly respond to environmental conditions to emerge from dormancy.