Melanization of a secondary sexual structure facilitates male mating success in Drosophila.

An integrative approach is used to identify a mechanism by which the fitness effects of Y chromosomes can manifest via trans effects on autosomal genes, through their effects on male courtship and other sexually selected traits.

Engineering male mosquitoes lacking the fruitless gene induces them to be attracted to humans.

The fossil record of abdominal modifications in male scorpionflies documents a relatively late evolution of oppressive behaviour toward a female during mating and suggest degree of sexual coercion greater in Eocene than in any other known scorpionfly, extinct or extant.

Male C. elegans die through two distinct mechanisms – mating-induced germline activation, and potent male pheromone toxicity – but the latter is unique to males of androdioecious species (made up of hermaphrodites and males).

A screen targeting RNA-associated proteins reveals that PSI regulates timeless alternative splicing and thus controls the period of Drosophila circadian behavior and its phase under temperature cycles.

In the brain of medaka fish, neuropeptide B acts directly downstream of estrogen in a female-specific but reversible manner to mediate female receptivity to male courtship.

In nematode worms, the length of the male refractory period–the time between matings–is regulated by multiple transmitters including dopamine, which both promotes ejaculation and reduces the activity of males post-copulation.

The first evidence is presented of a compound released by mated females that directly counteracts the effects of a seminal anti-aphrodisiac, but has no attractant properties in itself.

A novel developmental mechanism generates a previously undescribed pair of sex-specific proprioceptive neurons required for male-specific behaviour.