An atypical neuronal pacemaker mechanism triggers feeding motor pattern in Aplysia..

Pacemaker neurons are present in the preBötC circuitry of mouse embryos and their role in driving respiratory network activity changes during the critical period immediately before birth.

The most influential clocks within the Drosophila circadian clock neuron network form the fewest synapses within the network, and neurons that do not themselves contain molecular clocks mediate connections between those that do, suggesting a key role in timekeeping.

The neuromodulator substance P enhances mechanisms of recurrent excitation between breaths to control breathing frequency and stability.

The non-selective Na⁺ leak channel (NALCN) and the transient receptor potential 3 channel (TRPC3) cooperate in driving pacemaking in substantia nigra dopaminergic neurons.

Kinetic interactions between sodium channels and auxiliary factors create a molecular computational engine that can sense and regulate cellular excitability.

The phase of a neuron in an oscillatory network can remain independent of network frequency when synaptic input strength and peak phase are adjusted in a frequency-dependent manner.

The mechanism that couples two rhythmic circuits is maintained over a large temperature range although both circuits are temperature-sensitive.

Laser-ablating neurons of a single genetic class reveals that Dbx1-derived interneurons comprise core respiratory rhythmogenic and premotor circuits and provides quantitative cellular parameters that govern network functionality

The hyperpolarization-activated inward current plays an important role in regulating both smooth transitory responses and persistent increases in pyloric frequency during temperature fluctuations in Cancer borealis.