Selective synapse formation in a retinal motion-sensitive circuit is orchestrated by starburst amacrine cells, which use homotypic interactions to initiate formation of a dendritic scaffold that recruits projections from circuit partners.

Regulation of Pax6 expression through the α-enhancer fine-tunes amacrine cell subtype number, and consequently, the visual output of the retina.

Synapse-specific genetic manipulations show that distinct GABAergic inputs are differentially recruited to encode motion direction in the retina in a stimulus-dependent manner.

Experimental and computational models reveal how parallel 'core' mechanisms shape direction selectivity at the dendrites of starburst amacrine cells and ganglion cells in the mouse retina.

Heterogeneity in perceptual sensitivity of human cone-mediated vision across wavelength originates in the cone photoreceptors, where S cones exhibit distinct functional properties in comparison to L and M cones.

Mitochondria reach stable positions in dendrites during a critical developmental period of high motility.

A quantitative analysis of the connectivity between photoreceptors and bipolar cells in the mouse retina based on electron microscopy data yields exceptions from established rules of outer retinal connectivity.

The encoding of visual images by certain retinal ganglion cells is fundamentally altered in the context of eye-movement-like image transitions; the transitions trigger inhibitory interactions, which make these cells particularly sensitive to recurring images.

The convergence of two visual pathways at the level of retinal bipolar cells accounts for key features of ganglion cell responses.

A single class of neurons switches both its sensory detection and neurotransmission mechanisms to support transient or sustained physiological output.