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Dentate nucleus neurons can dynamically modulate their activity during a visual attention task, comprising not only sensorimotor but also cognitive attentional components.

Optogenetic activation of cortical GABAergic cells in macaques yields robust and reversible behavioral deficits.

Peripheral retinal input transiently amplifies information transmission from ganglion cells, dynamically allocating the resources of neural activity to times of expected high information content.

Perceptual decision making in monkeys can be manipulated by using optogenetics to inactivate functional subregions of visual cortex, but the brain has a capacity to compensate for this perturbation.

The encoding of visual images by certain retinal ganglion cells is fundamentally altered in the context of eye-movement-like image transitions; the transitions trigger inhibitory interactions, which make these cells particularly sensitive to recurring images.

Involuntary eye movements that keep images positioned over the center of the retina are synchronized with blinks to minimize disruption to vision.

Oculomotor circuits are always busy planning the next eye movement, and this explains why, when a visual target appears, some eye movements toward it are produced very quickly whereas others take a long time to prepare.

Midbrain neurons display attention-related modulation even in the absence of microsaccades, demonstrating that shifts of attention can be dissociated from the generation of microsaccades.

Sensory activity within the superior colliculus is read out by downstream motor structures and contributes to individual saccade kinematics with single spike precision.

Adaptation along multiple task-axes reveals choice traces at millisecond resolution in non-invasive recordings from human premotor cortex and provides evidence that theories of top-down control hold even after extensive experience.