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Optogenetically induced motor disruption can evoke plasticity in the mouse frontal cortex that compensates for the motor deficits, accompanying changes in inter-hemispheric motor representation.

Secondary somatosensory and visual thalamus monitor behavioral state, rather than movement, and may exist to alter cortical activity accordingly.

Electrophysiological recordings and optogenetic manipulations revealed the distinct roles that acetylcholine can have on the amygdala and prefrontal cortex in a Window of Opportunity Task.

The consolidation of newly acquired motor skills induces a functional reorganization of sequential information representations within secondary motor cortex, basal ganglia and hippocampus.

Preparatory states within the motor cortex play a critical role in selecting an appropriate action in response to sensory input, dependent on the context.

Transient expression of Nrp1 is required in postnatal stages for terminal arborization and innervation of secondary areas during the development of somatosensory callosal circuits.

Introducing a hominoid-specific gene into mice induces cortical folding.

A detailed physiological characterization of inputs to the motor thalamus adds nuance to the notion that information from the cerebellum and basal ganglia are segregated and provides a mechanism by which the basal ganglia can gate communication between cortical areas.

Noninvasive brain stimulation can artificially tag and retrieve human motor memories by altering the background activity patterns of the sensorimotor cortex.

The combination of intraneural microstimulation and 7T fMRI makes it possible to bridge the gap between first-order mechanoreceptive afferent input codes and their spatial, dynamic and perceptual representations in human cortex.