Behavioral and neurophysiological recordings in infant rats reveal that sleep and sensory experience influence neural activity in prefrontal cortex, mirroring similar findings in developing sensorimotor cortex.

Prediction of future input explains diverse neural tuning properties in sensory cortex.

Delay-period activity of anterior piriform cortex is important for working memory tasks requiring active maintenance and encodes the maintained information.

Local sensory signals in visual and frontal cortex play a causal role in task performance, while widespread dorsal cortical signals correlating with movement reflect processes that do not.

Local cortical sleep features arise subcortically due to heterogeneous burst discharge in neurons of a sleep rhythm pacemaker previously thought to act uniformly.

Early in development, before neurons in primary motor cortex are involved in motor control, they undergo a rapid transition in how they process sensory information following sleep and wake movements.

Decision-makers are able to intentionally control neural excitability to strategically bias sensory evidence accumulation towards the decision bound that maximizes reward within a given ecological context.

The complement pathway can contribute to corneal sensation loss and nerve damage during ocular surface inflammation.

Sensory-evidence accumulation is a distributed cortical computation, but frontal cortical areas contribute to accumulation on longer timescales than posterior cortical areas.

Deactivation of one side of the auditory midbrain while recording in the other shows that the two sides cooperate in processing frequency and in enhancing the encoding of sound level.