Aging reduces the potassium M current in sympathetic motor neurons, resulting in a hyperactive phenotype that can be reversed by pharmacological activation of these channels.

In vivo analysis of endogenously tagged Ca²⁺ channel subunits reveals unexpected differences in subunit composition and synapse-specific relationships between channel abundance and synaptic strength.

The human cerebellum shows fMRI activity for many tasks – however, the inputs from the neocortex are upregulated in a task-dependent manner when cerebellar computation is required.