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The higher order thalamus encodes and influences correct action during learning and performance in a sensory-based goal-directed behavior.

Secondary somatosensory and visual thalamus monitor behavioral state, rather than movement, and may exist to alter cortical activity accordingly.

Dual site recordings of mouse anterodorsal thalamic nucleus and retrosplenial cortex reveal near 0-ms lag between the head direction representations of the two regions after cue rotations and correlated drift in darkness.

Salient or behaviorally relevant stimuli, regardless of their modality, robustly activate both higher-order somatosensory and visual thalamic nuclei.

Understanding how general anesthesia changes neural dynamics in the cortex and thalamus can lead to its safer use and shed light on the nature of consciousness.

A multi-scale integration of experimental and computational approaches shows how a non-linear dependence of T-type calcium channel gating on GABA_B receptor activity regulates thalamic network oscillations.

By differentially modulating the two major excitatory inputs to the striatum, mu- and delta-opioid receptors regulate the balance between thalamic and cortical inputs to the striatum.

The experience-dependent representation of time interval in the seconds-to-minutes range occurs as early as at the thalamic level in the brain.

Lesions to the anterior-medio-dorsal thalamus cause widespread behavioral impairments across multiple cognitive domains, suggesting that thalamic hubs are critical for interconnecting diverse cognitive processes.

Retinal ganglion cell axons release the morphogen sonic hedgehog which signals through astrocytes to induce the migration of inhibitory interneurons into the developing visual thalamus.