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Engineered anion-conducting channelrhodopsins with enhanced red-light sensitivity and accelerated kinetics enable precise, low-intensity optical silencing of neurons, advancing optogenetic control in neuroscience research.

Two-module networks, combining sensory efficient coding and attractor dynamics for memory maintenance, effectively explain working memory error patterns, highlighting the role of sensory-memory interactions in continuously shaping memory representations.

The autobiographical memory deficits seen in aphantasia are reflected by altered activation and connectivity patterns of the hippocampus and occipital cortex, corroborating the strong link between memory and mental imagery.

Neuronal loss can be studied by passing electrical current through the same electrodes used for neuron measurement without disrupting recording ability.