A new proof of principle highlights the requirements and limitations associated with using barcoded versions of the rabies virus to dissect neural networks.
Decreasing the levels of a protein that is overproduced in Alzheimer’s disease restores neuronal connectivity and memory in a mouse model of the condition.
Experiments in mouse cells reveal that adolescent idiopathic scoliosis may be triggered by estrogen signaling in the cartilage cells of the spine, which could partly explain the female bias in the condition.