1. Immunology and Inflammation
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Correction: The transforming growth factor beta signaling pathway is critical for the formation of CD4 T follicular helper cells and isotype-switched antibody responses in the lung mucosa

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Cite this article as: eLife 2015;4:e06900 doi: 10.7554/eLife.06900

Main text

Marshall HD, Ray JP, Laidlaw BJ, Zhang N, Gawande D, Staron MM, Craft J, Kaech SM. 2015. The transforming growth factor beta signaling pathway is critical for the formation of CD4 T follicular helper cells and isotype-switched antibody responses in the lung mucosa. eLife 4:e04851. doi: 10.7554/eLife.04851

Published 8 January 2015

TGF-β was incorrectly spelt out as ‘tumor growth factor beta’ both in the title and in the Digest. The correct title is: The transforming growth factor beta signaling pathway is critical for the formation of CD4 T follicular helper cells and isotype-switched antibody responses in the lung mucosa. The title and Digest have been corrected accordingly.

Article and author information

Author details

  1. Heather D Marshall

    Competing interests
    The authors declare that no competing interests exist.
  2. John P Ray

    Competing interests
    The authors declare that no competing interests exist.
  3. Brian J Laidlaw

    Competing interests
    The authors declare that no competing interests exist.
  4. Nianzhi Zhang

    Competing interests
    The authors declare that no competing interests exist.
  5. Dipika Gawande

    Competing interests
    The authors declare that no competing interests exist.
  6. Matthew M Staron

    Competing interests
    The authors declare that no competing interests exist.
  7. Joe Craft

    Competing interests
    The authors declare that no competing interests exist.
  8. Susan M Kaech

    Competing interests
    The authors declare that no competing interests exist.

Publication history

  1. Version of Record published: February 12, 2015 (version 1)

Copyright

© 2015, Marshall et al.

This article is distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use and redistribution provided that the original author and source are credited.

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