A study in a rat model of temporal lobe epilepsy has identified sodium selenate as a promising drug candidate that could reverse progression of the condition.
Pathogenic forms of the ANT1 protein lead to harmful consequences in yeast and mice by clogging the channels that allow mitochondria to import proteins.
A new mouse model allows accurate detection of oligodendroglia that produce immune proteins, paving the way for more detailed studies of the role these cells play in multiple sclerosis.