Tmc7 expression is confined to the testis, and its deletion leads to male infertility.
(a) Heatmap analysis of the expression of Tmc family members at embryonic day (E)10–E18 and different postnatal days (P0, P3, PD14, PD28, and PD63) in mouse testis using the Evo-devo mammalian organ database (http://apps.Kaessmannlab.org/evodevoapp/). (b) The analysis of Tmc7 expression in different tissues using the Mouse Cell Atlas database (https://bis.zju.edu.cn/MCA/). (c, d) T-Distributed Stochastic Neighbor Embedding (t-SNE) plot and violin plot showing the expression of Tmc7 in different mouse germ cells using the Male Health Atlas (http://malehealthatlas.cn/). SSC1-3: Spermatogonial stem cells I-III; Progenitor: spermatogonia progenitor cell; A1–4: A1–4 spermatogonia; In: Intermediate spermatogonia; B: Type B spermatogonia; PI: Preleptotene; L: Leptotene; Z: Zygotene; P: pachytene; D: Diplotene; M: Metaphase; RS: Round spermatid. E: Elongating spermatid. (e) The expression of Tmc7 in different adult mouse tissues was measured by RT-PCR, including the heart, liver, spleen, lung, kidney, brain, ovary, and testis. Gapdh was used as the control. (f) The expression of Tmc7 at different postnatal times (PD1, PD7, PD9, PD12, PD24, PD33, and 2 months) was measured by RT-PCR. Gapdh was used as the control. (g) Schematic illustrating the generation of Tmc7−/− mice using CRISPR/Cas9. (h) Testis from 9-week-old WT and Tmc7−/− mice. (i) Testicular weights of 9-week-old WT and Tmc7−/− mice. Six adult mice of each genotype were used. (j) Testis/body weight ratio of 9-week-old WT and Tmc7−/− mice. Six mice of each genotype were used. Images are representative of at least three independent experiments. The results are presented as the mean ± S.D., and two-sided unpaired Student’s t-tests were used to calculate the P-values (*P < 0.05, **P < 0.01, ***P < 0.001).