1. Neuroscience
  2. Structural Biology and Molecular Biophysics
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Disease-associated mutations in the human TRPM3 render the channel overactive via two distinct mechanisms

  1. Siyuan Zhao  Is a corresponding author
  2. Yevgen Yudin
  3. Tibor Rohacs  Is a corresponding author
  1. New Jersey Medical School, Rutgers University, United States
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Cite this article as: eLife 2020;9:e55634 doi: 10.7554/eLife.55634
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