Interhemispheric inhibition can be manipulated by directly and bidirectionally modulating the bilateral sensorimotor excitabilities in a spatially bivariate Brain-Computer Interface-based neurofeedback paradigm.

A neural gating mechanism in the external cuneate nucleus of the rat brain is engaged during wake movements and disengaged during sleep-related twitches.

Burst-like neural activity in the β-frequency band conveys inhibitory commands within long-proposed cortico-subcortical networks for motor inhibition, with inhibitory activity in STN preceding thalamic activity, which has strong implications for movement disorders marked by abnormalities in β-bursting.

The ability to quickly re-acquire a previously lost motor skill is associated with lasting synaptic changes in the brain circuit that controls that motor skill.

Response inhibition is initiated by the right inferior frontal gyrus (rIFG), and stopping performance is predicted by beta-band power as well as beta-band connectivity between rIFG and pre-supplementary motor area.

Hierarchical Bayesian modeling of stop-signal task performance in humans with and without lesions to the right inferior gyrus reveals that rather than implementing inhibitory control, this brain region appears to be responsible for detecting the need to inhibit an action.

Tonic disinhibition of left motor cortex during prism adaptation enhanced consolidation of sensorimotor and cognitive prism after effects, causing lasting clinical gains in three patient cases with chronic treatment-resistant visual neglect.

Non-invasive disinhibition of the oculomotor system shows that ongoing preparatory activity in the superior colliculus has movement-generating potential and need not rise to threshold in order to produce a saccade.

The selective effect of local inhibition on diffuse patterns of brain connectivity can be accounted for by an intrinsic hierarchical ordering of cortical timescales.

The brain inhibits the formation of new memories just after waking to safeguard the ongoing stabilization of existing memories during sleep.