A new method for inducing, measuring and modelling visual hallucinations in healthy people reveals that hallucinations share common underlying mechanisms with normal sensory perception.

fMRI evidence for distinct hierarchical alterations in intrinsic neural timescales for different positive symptoms of schizophrenia support hierarchical perceptual-inference models of psychosis and suggest local increases in excitation-inhibition ratio.

Autistic traits are associated with weaker influence of prior expectations in visual perception, which is due to more precise sensory representations.

Psychedelics induce a pattern of cortical activity very similar to the one observed during visual perception, despite participants had shut eyes.

Multiple non-redundant features of non-rapid eye movement sleep are altered in schizophrenia and largely independent of waking electrophysiological abnormalities, supporting the promise of neuropsychiatric disease biomarkers based on a precise dissection of the sleep.

Sensory stimulation produces stochastic population responses in the cortex that are governed by layer 6 cortico-reticular projections that modulate thalamic synchrony.

Cell type-specific effects of antipsychotic drugs in cortex could provide a basis for a functional screen of antipsychotic efficacy.

Targeted optogenetic activation of small ensembles of neurons is sufficient to trigger a behavioral report while recruiting matched network suppression, suggesting exquisite sensitivity despite network mechanisms that maintain sparseness.

A citizen science approach to research has shown that the improvements in mood and cognition associated with psychedelic microdosing are likely due to a placebo effect.

A cortical model inspired by cognitive theories and deep learning demonstrates how the combination of wakefulness, non-rapid eye movement (NREM), and REM dreams leads to the emergence of robust and semantically organized neuronal representations in higher cortical areas.