The systematic field testing of excreta from Australian native possums for the bacterial pathogen Mycobacterium ulcerans can be used to build statistical models that predict the regions in southeast Australia where humans will subsequently get Buruli ulcer.

The secreted toxin mycolactone has severe effects on the cells lining blood vessels that will play a critical role in the development of swelling in Buruli ulcer disease.

Different transmission routes of Mycobacterium ulcerans to aquatic organisms may prevail depending on local physico-chemical conditions.

A better understanding of the mechanisms underpinning the growth of mycobacteria could help identify targets for new antibiotics.

The structure of the Mycobacterium smegmatis CIII₂CIV₂ respiratory supercomplex with telacebec (Q203) bound shows how this tuberculosis drug candidate blocks respiration in mycobacteria.