Browse our Research Articles

Telomere dysfunction in iPSC-derived type II alveolar epithelial cells causes senescence and gene expression changes including Wnt-related changes that GSK3 inhibition can rescue, providing insight into pulmonary fibrosis pathogenesis.

Human intracranial electroencephalographic recordings reveal the electrophysiological properties and hierarchical organization of spontaneous neuronal oscillations in the human insula and show that these oscillations are traveling waves, thus providing new insights into intrainsular and interinsular communication.

Single unit recordings in monkeys and biophysical modelling demonstrate that local inhibitory-controlled metastable neural states specify the temporal organization of cognitive functions in frontal areas.

The more semantically related a later experience is to an earlier one (along multiple dimensions), the more likely humans are to think back to and strengthen the memory of the earlier experience and mentally link the two experiences.

Metabolomics data reveals unique metabolic insights into how cavefish adapt to nutrient limited environments while staying healthy.

While performing learned movements at different speeds, motor cortex population activity can be understood based on the need to generate muscle activity via smooth, well-behaved dynamics.

Because voltage-gated KCNQ2 channels are central to physiological and pathophysiological events, understanding how disease-causing mutations in different channel regions disrupt function will help future development of mutation-specific antiepileptic therapies.

Analysis of performance on a novel behavioural paradigm investigating active information gathering reveals that subjective cognitive impairment is associated with hyperreactivity to uncertainty and links this mechanism to the affective burden in the condition and heightened insular-hippocampal connectivity.

Optogenetic and electrical stimulation of primary visual cortex neurons reveals synaptic and cell-intrinsic mechanisms that underlie neuronal ensemble formation.

4,6-Sulfation of chondroitin sulfate proteoglycans prevents sympathetic nerve regeneration into the infarct after myocardial ischemia/reperfusion, and reducing this sulfation promotes nerve regeneration and decreases arrhythmia susceptibility.