Browse our latest Neuroscience articles

Photobleaching and recovery of a fluorescent glutamate sensor enable demonstration of the distinct spatial architectures of spontaneous versus evoked forms of glutamatergic neurotransmission within single synapses.

The explanation of why older people show increased behavioral variability despite having decreased neural signal variability lies in the link between the dynamics of the ongoing neural signal and the trial-by-trial variability of neural evoked responses.

Functional imaging (fMRI) across four mammalian species maps the brain areas engaging in burst-suppression activity during anesthesia, and uncovers differences between primates and rodents.

Fluorescent glutamate sensors shed light on the microscopic organization underlining spontaneous neurotransmission.

A standardized comparison of contractile force among in vitro muscle tissues across different platforms requires proper normalization with the contractile area and complementary morphological, maturation, and functional analysis to reduce variability and boost a new generation of engineered constructs.

Colored surfaces induce gamma oscillations in human early visual cortex that are equally strong for red and green colors when L-M cone contrast is controlled for.

A comprehensive excitatory projectome of the ventro-lateral prefrontal cortex in macaques was generated by using viral tracing integrated with serial two-photon tomography and diffusion tractography, thus providing new evidence of rejecting the monosynaptic connection of inferior fronto-occipital fasciculus in monkeys.

The anterior insula cortex is a critical neural substrate of context-induced relapse of nicotine-seeking.

The hypothalamic parasubthalamic nucleus can be subdivided into two distinct populations, one of which decreases food consumption and is necessary for the full appetite-suppressing effects of anorexigenic hormones.

The dorsal medial prefrontal cortex and basolateral amygdala exhibit social behavior-relevant neuronal oscillations, representing unified pathophysiological mechanisms underlying social behavioral deficits.