Browse our Research Articles

Recurrent spiking networks that process input stimuli with optimal efficiency have key emerging properties that are similar to those of biological neural networks.

The intrinsic factor of cell senescence, PCBP2, was identified by unlabeled quantitative proteomics, and its biological role in in vitro replication senescence of human bone marrow mesenchymal stromal cells was characterized in vitro.

Multi-dimensional social relationships dynamically shape attentional biases toward in-group and out-group conspecifics in monkeys, with oxytocin modulating these interactions to reveal neurobiological links between social networks and visual information processing.

Plectin drives hepatocellular carcinoma progression and metastasis through cytoskeletal reorganization and oncogenic signaling, making it a promising therapeutic target for overcoming therapy resistance.

UNC-6/Netrin functions both as a short-range adhesive cue and a gradient to guide axons in vivo.

Assessment of transfer entropy is useful for network analysis of cell signaling network providing information on the network topology and action points of genetic mutations and drug effect, thus will be applicable to molecular pharmacology and pathology.

The stem cell signal Msi2 plays a dual role in lung cancer, acting as a signal that is not only essential for initiation, but also for propagation of the tumor.

The nucleus reuniens of the thalamus play a central role in synchronizing the mPFC-hippocampal network during sleep and integrates converging activity from hippocampal and mPFC oscillations.

The visual system of infants has adult-like properties, and these properties can be revealed at an individual level by having infants watch movies during functional magnetic resonance imaging.

Molecular, biophysical, and computational analysis reveals insights on the mechanism of association of birnavirus VP3 with endosomes and its role in the viral life cycle, representing a substantial contribution toward understanding the replication strategy of these 'non-canonical' viruses.