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An inferred mechanistic model that connects sequence (genotype) to function (constitutive gene expression phenotype) for any random sequence in Escherichia coli reveals the structure of constitutive promoters and how they evolve.

The evolution of division of labour with non-accelerating returns from specialisation can occur for several reasons, including but not limited to topologically constrained group structures, but there must always be a group efficiency benefit and a coordination mechanism is required.

A small molecule compound Dwn1, identified through high-throughput screening using Npas2 as the molecular target, enhances wound healing in a murine incisional wound model.

A machine learning model could accurately estimate daily salt consumption in the general population and can be applied to countries lacking urine sample to compute the mean salt consumption at the population level.

The perception of endogenous RALF signaling peptides modulates the plasma membrane nanoscale organization of receptor kinases to regulate plant immune signaling.

The Arabidopsis N6-methyladenosine (m⁶A) binding proteins ECT2 and ECT3 act redundantly in the cytoplasm to regulate m⁶A-containing mRNAs whose abundances are reduced in ect2/ect3/ect4 knockout plants although the locations of their polyadenylation sites remain unaffected, contrary to currently held beliefs.

The Arabidopsis N6-methyladenosine (m⁶A)-binding protein ECT2 binds to m⁶A in the canonical RR[m⁶A]CH motif, not a plant-specific motif as previously thought, and its intrinsically disordered region contributes to mRNA binding via contacts with U-rich motifs.

A mechanism whereby aminoglycosides-mediated SOS induction in Escherichia coli also promote high-level fluoroquinolone resistance.

The fly connectome reveals dedicated cell types for processing specific kinds of visual information such as color or skylight polarization.

Overexpression of interferon-stimulated genes revealed a role for RARRES3 in restricting growth of Toxoplasma gondii by inducing early egress from human cells.